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The initial configuration of the Oracle Exadata Database Machine is typically performed by Oracle ACS upon delivery and installation You will only need to perform routine ongoing management of its hardware and software components The Oracle database and the Oracle Grid infrastructure components of the Database Machine are treated like any other Oracle install on a Linux or a Solaris platform, and are maintained and monitored just like you would maintain and monitor any other Oracle RAC system using ASM The Exadata Storage Servers require minimal management since the bulk of administration tasks are performed during the initial setup phase

overlying skin may be reddened and slightly swollen Trauma may be recalled as the initiating factor, but as the months pass, other muscles not injured in any recognizable way become similarly involved At rst, radiographs reveal no important changes, but within 6 to 12 months, calcium deposits are observed, and one can feel stony-hard masses within the muscles As the disease advances, limitation of movement and deformities become increasingly evident Calci ed bridges between adjacent muscles and across joints lead to rigidity of the spine, jaw, and limbs; scoliosis; and limited expansion of the thorax Ultimately, the patient is virtually changed to stone The principal problem in diagnosis is to differentiate generalized myositis ossi cans from calcinosis universalis The latter usually occurs in relation to scleroderma or polymyositis and is characterized by calcium deposits in the skin, subcutaneous tissues, and connective tissue sheaths around the muscles; in myositis ossi cans, there is actual bone formation within the muscles The pathologic data are often too meager to justify this sharp distinction The prolonged ingestion of large doses of vitamin D may also result in the deposition of masses of calcium salts around muscles, joints, and subcutaneous tissue Calci c deposits, perhaps true ossi cation, may occur in the soft tissues around the hips and knees of paraplegics and rarely following a hemiplegia ( paralytic myositis ossi cans ) Myositis ossi cans may undergo spontaneous remissions and may stabilize for many years, during which the patient is capable of adequate function In other cases, progression leads to marked debilitation and respiratory embarrassment, the nal illness often being a terminal pneumonia or other infection The molecular basis for myositis ossi cans is not known, but it has been suggested that one causative defect is the overexpression of bone morophogenic protein In mice, the forced expression of this protein induces heterotopic bone formation It is likely that the primary problem arises either because of inappropriate expression of one element of the protein or of excessive binding between signaling proteins and their receptors (Glaser et al) Treatment The administration of a diphosphonate [ethane-1-hydroxy-1,1-diphosphate (EHDP), 10 to 20 mg/kg orally], a compound that inhibits the deposition of calcium phosphate, has been said to cause regression of new swellings and to prevent calci cation (Russell et al) Some of the calcium deposits in calcinosis universalis have receded in response to prednisone, and because of the unclear relationship of this disease to generalized myositis ossi cans, it is probably advisable to try this form of therapy as well Excision of bony deposits may be undertaken if it is certain that they are the cause of particular disabilities.

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to eject the disk and insert another If the problem goes away, the disk, not the drive, is the source of the problem If you must have the information on The floppy disk, rather than a particular disk that s giving you trouble, try the drive, is the cause of most floppy errors gently pulling the metal cover back and letting it snap back into place a few times, then reinserting the disk in the drive and trying to access the data again You can also try to use the disk in a different drive

ABERFELD DC, HINTERBUCHNER LP, SCHNEIDER M: Myotonia, dwarfism, diffuse bone disease, and unusual ocular and facial abnormalities (a new syndrome) Brain 88:313, 1965 BANKER BQ, ENGEL AG: Other in ammatory myopathies, in Engel AG, Franzini-Armstrong C (eds): Myology, 3rd ed New York, McGraw-Hill, 2004, pp 1445 1472 BANKER BQ, CHESTER CS: Infarction of thigh muscle in the diabetic patient Neurology 23:667, 1973 BARKER RA, REEVES T, THOM M, et al: Review of 23 patients affected by the stiff man syndrome: Clinical subdivision into stiff trunk (man) syndrome, stiff limb syndrome, and progressive encephalomyelitis with rigidity J Neurol Neurosurg Psychiatry 65:633, 1998 BLEXRUD MD, WINDEBANK AJ, DAUBE JR: Long term follow-up of 121 patients with benign fasciculations Neurology 34:622, 1993 BRODY IA: Muscle contracture induced by exercise: A syndrome attributable to decreased relaxing factor N Engl J Med 281:187, 1969 BROWN P, ROTHWELL PJ, MARSDEN CD: The stiff leg syndrome J Neurol Neurosurg Psychiatry 62:31, 1997

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