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Characterized by elevation of the ST segment in leads V1 V3, right bundle branch block and susceptibility to arrhythmias. Thought to be more common in the Far East (known as sudden unexplained nocturnal death syndrome) (Priori et al. 2003) There are several patterns, which are characterized by varying degrees of myocardial hypertrophy, gross disorganization of muscle bundles and cardiac microstructural architecture which predispose to arrhythmias and sudden death (Firoozi et al. 2002)

Table 19.4 Conditions associated with sudden cardiac death in athletes and their genetic component. Condition Long QT syndrome Clinical implications QT interval prolongation associated with ventricular tachyarrhythmias, syncope and sudden cardiac death Genetic in uence Both autosomal dominant (Romano Ward) and recessive forms (Jervell and Lange Nielsen) have been described (Priori et al. 2003). A number of genetic loci and genes have been identi ed and associated with the syndrome: KCNQ1 gene on chromosome 11 (LQT1) (Wang et al. 1996) KCNH2 gene on chromosome 7 (LQT2) (Curran et al. 1995) SCN5A gene on chromosome 3 (LQT3) (Wang et al. 1995) KCNE1 and KCNE2 genes on chromosome 21 (LQT 5 & 6) (Splawski et al. 1997) These genes code for ion-channel subunits that in uence cardiac myocyte excitability (Priori et al. 2003) Genetic testing is only successful in 55 65% of affected patients (Splawski et al. 2000; Priori et al. 2003) suggestive of the presence of other relevant genes The pattern of inheritance appears to mainly be autosomal dominant (Leenhardt et al. 1995) Mutations of the cardiac ryanodine receptor (RyR2) gene identi ed onchromosome 1 have been associated with the syndrome (Priori et al. 2001) which is involved in the release of Ca2+ during the Phase 2 of the action potential A less common recessive form has been linked to the CASQ2 gene on chromosome 1 which encodes for calsequestrin, which serves as a Ca2+ reservoir in the heart and is linked to RYR2 and compatible with the less common recessive form of the syndrome (Lahat et al. 2001) Associated with mutations in the cardiac sodium channel gene (SCN5A), but is likely that other mutations contribute to the syndrome (Chen et al. 1998; Priori et al. 2002, 2003)

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In this case, the first thing on the line is a process ID. This is because W32SVSPY couldn't extract the process name from the Win16 Task Database (TDB) that Windows 95 creates for each process. This usually happens only during application startup, before KERNEL32.DLL has

Marco had his own tense moment. He was waiting for a winning hand when a pit boss he hadn t noticed stepped up to his shoulder. You doubled up to five thousand dollars that s some luck, he said, surprised. An old woman at the next machine piped up in a smoker s raspy sandpaper voice, It ... wasn t ... luck. The pit boss stiffened, his suspicions aroused. It was balls, she cawed. The pit boss smiled and walked away. Over a period of about three years, the guys alternated between taking legitimate consulting jobs to keep up their skills and contacts, and skipping out now and then to line their pockets at the video poker machines. They also bought two additional machines, including the most widely used video poker model, and continued to update their software. On their trips, the three team members who traveled would head out to different casinos, not all go as a pack, Alex said. We did that once or twice, but it was stupid. Though they had an agreement to let each other know what they were up to, occasionally one would slip away to one of the gambling cities without telling the others. But they confined their play to casinos, never playing in places like 7-Elevens or supermarkets because they tend to have very low payouts.

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